Titlebook: Introduction to Gastrointestinal Diseases Vol. 2; Jakub Fichna Book 2017 Springer International Publishing AG 2017 Gastrointestinal tract.

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Pathophysiology and Risk Factors in Peptic Ulcer Diseasenoxious and protective factors in pathophysiology of PUD are discussed. The impact of obesity, alcohol consumption, smoking, as well as genetic factors and use of NSAIDs are also presented. Nowadays, the effective therapies based on inhibition of gastric acid production and . eradication provide a g

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Clinical Features in Peptic Ulcer Diseasef symptoms of PUD will be reviewed. Authors paid special attention to the most frequent symptom of PUD which is abdominal pain..If diagnosis of PUD is delayed or management is improper, complications such as gastrointestinal bleeding and perforation of gastric outlet obstruction (GOO) may occur. In

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Pharmacological Treatment of Peptic Ulcer Diseaseiology of PUD is relatively well understood, hence there are several specific treatment options for this disorder aiming at various pharmacological targets located in the GI tract. In this chapter we provide an overview on pharmacological targets as well as drugs that are currently available for PUD

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Surgical Treatment of Peptic Ulcer Disease2 blockers and proton pump inhibitors over the last several decades and the recognition that treatment for . infection can eliminate most ulcer recurrences have essentially eliminated the need for elective surgery. However, complications related to PUD continue to occur and include bleeding, perfora

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Patient’s Guide: Diet and Lifestyle in Peptic Ulcer Disease elaborate on how dietary modulations and lifestyle habits can affect the course of . infection. We depict the role of several dietary components that may protect against the pathological consequences of . infection, and thus improve patient’s eradication therapy. Considering . as a strong and estab

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Pathogenesis of Colorectal Cancermolecular genetics and epigenetics of colorectal cancer have led to novel insights into its pathogenesis. The results show that both pathogenic mechanisms and the influence of the etiological factors create a complex and multifarious network of events. Somatic and germline mutations, alongside with