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Titlebook: Interstitial Fibrosis in Heart Failure; Francisco J. Villarreal Book 2005 Springer-Verlag New York 2005 gene therapy.heart.heart failure.m

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发表于 2025-3-21 17:12:56 | 显示全部楼层 |阅读模式
书目名称Interstitial Fibrosis in Heart Failure
编辑Francisco J. Villarreal
视频video
概述Focused volume on cardiac extracellular matrix structure.This topic normally covered as a chapter in larger heart failure texts.Provides more in-depth information.There is currently no direct competit
丛书名称Developments in Cardiovascular Medicine
图书封面Titlebook: Interstitial Fibrosis in Heart Failure;  Francisco J. Villarreal Book 2005 Springer-Verlag New York 2005 gene therapy.heart.heart failure.m
描述.Interstitial Fibrosis in Heart Failure., edited by Francisco J. Villarreal, M.D., Ph.D., provides a timely and integrative review of the basics of cardiac extracellular matrix structure. Topics covered include how cardiac remodeling influences its disposition, abundance and function; possible non-invasive techniques for diagnosis; and potential drug-based or molecular therapeutic strategies that may interrupt or even reverse the course of the development of cardiac fibrosis...This resource for both clinicians and scientists aims to cover state-of-the-art findings relevant to cellular and molecular processes underlying cardiac fibrosis including basic elements of structure, function, diagnosis and treatment..
出版日期Book 2005
关键词gene therapy; heart; heart failure; myocardial infarction; physiology
版次1
doihttps://doi.org/10.1007/b99419
isbn_softcover978-1-4419-1983-0
isbn_ebook978-0-387-22825-9Series ISSN 0166-9842
issn_series 0166-9842
copyrightSpringer-Verlag New York 2005
The information of publication is updating

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Pharmacology of G-Protein-linked Signaling in Cardiac Fibroblastscardiovascular diseases differ from “normal” CFs. Actions of hormones are likely much more complex . than ., as the example of . agonists makes clear. In addition, cross-talk amongst signaling pathways when cells are stimulated by multiple agonists complicates the picture and the overall responses.
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Ultrasonic Characterization of the Myocardiumce of myocardial fibrosis and how these measurements lay the foundation for the continuing development of clinically applicable noninvasive ultrasonic assessment techniques. Several studies were cited which show how ultrasonic propagation and scattering are directly related to fundamental physical p
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Cardiac Mast Cells as Mediators of Ventricular Remodelingdiac chamber distension there is a release of atrial natriuretic peptide, which is known to cause mast cell degranulation (Figure 2). Secreted . tryptase and chymase are all capable of activating matrix metalloproteinases, which in turn are responsible for fibrillar collagen degradation. Since one o
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Biological and Functional Effects of Chronic Mechanical Support Induced by Left Ventricular Assist Dtechnology improves and we are able to avoid long-term mechanical failures and reduce the complications associated with the implantation of these devices. However, what has become clear is the failing heart, which appears to be “endstage,” may have the remarkable ability to recover at least at the c
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