Titlebook: Interferons; Biological Activitie Carlo Aul,Wolfgang Schneider Conference proceedings 1997 Springer-Verlag Berlin Heidelberg 1997 Autoimmun

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书目名称Interferons影响因子(影响力)




书目名称Interferons影响因子(影响力)学科排名




书目名称Interferons网络公开度




书目名称Interferons网络公开度学科排名




书目名称Interferons被引频次




书目名称Interferons被引频次学科排名




书目名称Interferons年度引用




书目名称Interferons年度引用学科排名




书目名称Interferons读者反馈




书目名称Interferons读者反馈学科排名

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acquisition

,The Role of Endogenous Interferon-α in HIV Infection and Autoimmune Diseases—An Overview,In the course of HIV infection, the role of interferon-α (IFN-α) seems to be rather paradoxical: “It is possible to view alpha-IFN as either a therapeutical agent or as a major contributor to the pathology associated with AIDS.” (S. Krown, 1992).

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,Use of Interferon-α in the Treatment of Chronic Myelogenous Leukemia,Chronic myelogenous leukemia (CML) is a clonal disease of hematopoietic stem cells [1]. It is characterized by an excessive increase in granulopoiesis and frequently also in megakaryopoiesis. The diagnostic criteria may vary slightly between authors and countries but in general include:

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,Distribution of Interferon-α2 Genes in Humans,ells and the nature of the infecting virus. Over the past 20 years it has become clear that there are many forms of IFNs produced. They can be grouped into four IFN families based upon their similarities and differences: alpha, beta, gamma, and omega IFNs. As shown in Table 1, only the alpha-interfe

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Interferon Therapy of Neuroendocrine Tumours,res and, not least, improved treatment, the number of cases has increased during the last two decades. The first case was described already in 1902 by Oberndorfer, who introduced the term . for a less malignant neoplasm. The incidence of patients with clinically significant carcinoid tumours is 0.5/

minion

Interferon Therapy of Essential Thrombocythemia and Polycythemia Vera,ineage bone marrow hyperplasia, whereas in essential thrombocythemia (ET), proliferation of the megakaryocytic lineage prevails. Both diseases run a relatively benign clinical course, and survival is almost normal. Morbidity and mortality result primarily from vascular complications, i.e., thromboem