Titlebook: Inflammatory Reaction; H. Z. Movat (Professor Dr.) Conference proceedings 1979 Springer-Verlag Berlin Heidelberg 1979 Entzündung.Reaction.

防御

The Role of Prostaglandins in Inflammation, can substitute for arachidonate in cell membranes, but apparently cannot prevent the deficiency. After . in 1935 had studied the polar fatty acids extracted from seminal plasma and termed them prostaglandins, . (1964) and . et al. (1964) simultaneously introduced the concept that PGs were derived f

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The Kinin System and Its Relations to Other Systems,reported by . (1958) and . et al. (1957), respectively. Bradykinin is cleaved from kininogen by plasma kallikrein. Kallikrein is generated from prekallikrein by activated factor XII (XIIa), the substance in plasma that becomes activated during contact with a negatively charged surface (see . et al., 1977; . et al., 1977; ., 1978, 1979a) (Fig. 1).

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Hyperemia, Stasis, and Increase in Vascular Permeability: New Methods for Their Quantitation,sion the redness due to hyperemia will disappear with death and cessation of blood flow, but the redness due to hemorrhage or stasis will persist. The swelling of inflammation is due to exudation of plasma into the tissues resulting from hyperpermeability of vessels of the microcirculation.

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Genome

Kinetics of the Inflammatory Response in Regional Lymph,hatic ducts. For the most part, lymph is composed of blood plasma containing approximately one-half the concentration of protein found in the blood. In addition, it contains free-floating mononuclear cells, particularly lymphocytes, but generally not red blood cells, granulocytes (PMN), or platelets.

AGOG

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Calculus

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