Titlebook: Inflammatory Mediators; Congress Proceedings G. A. Higgs,T. J. Williams Conference proceedings 1985 Palgrave Macmillan, a division of Macmi

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Neurogenic Inflammation,reactions in the periphery (1–3). Antidromic electrical stimulation as well as stimulation by chemical irritants or noxious heat lead to vasodilatation and plasma protein extravasation (4–6). These responses, which are also called “neurogenic inflammation” (see 7,8) are assumed to be caused by a med

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The Role of the Complement-derived Polypeptide C5a in Inflammatory Reactions,tentially harmful agent within the tissue, chemical messages are sent to the microvascular bed. The purpose of these messages is to promote a change in the local microenvironment such that plasma components and selected blood cells accumulate at the inflammatory focus. The functional role of this ac

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Phorbol Myristate Acetate and Platelet Activating Factor Promote Synthesis of Complement by Macrophgenerate and release the complement proteins C1 (and its subcomponents Clq, Cls), C2, C4, C3, C5, B, D, P, i.e. all components required for assembly of the classical or alternative pathway convertase. Complement synthesis and secretion by macrophages is governed by a range of factors, e.g. genetic r

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Production and Regulation of Human T Lymphocyte Chemotactic Factor (LCF),hanism of leukocyte locomotion and factors with the potential specifically to attract these cells to inflammatory foci .. The majority of research in this area has centred on the chemotactic response of neutrophils (PMN) and to a lesser extent on the response of monocytes and macrophages. Various na

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