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Titlebook: Inflammation-Associated Depression: Evidence, Mechanisms and Implications; Robert Dantzer,Lucile Capuron Book 2017 Springer International

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发表于 2025-3-21 19:43:30 | 显示全部楼层 |阅读模式
书目名称Inflammation-Associated Depression: Evidence, Mechanisms and Implications
编辑Robert Dantzer,Lucile Capuron
视频video
概述Reviews exhaustively the key recent research into the relationship between inflammation and depression.Covers the preclinical aspects of this research and its clinical implications.Broadens our unders
丛书名称Current Topics in Behavioral Neurosciences
图书封面Titlebook: Inflammation-Associated Depression: Evidence, Mechanisms and Implications;  Robert Dantzer,Lucile Capuron Book 2017 Springer International
描述Inflammation has invaded the field of psychiatry. The finding that cytokines are elevated in various affective and psychotic disorders brings to the forefront the necessity of identifying the precise research domain criteria (RDoCs) that inflammation is responsible for. This task is certainly the most advanced in major depressive disorders. The reason is that a dearth of clinical and preclinical studies has demonstrated that inflammation can cause symptoms of depression and conversely, cytokine antagonists can attenuate symptoms of depression in medical and psychiatric patients with chronic low grade inflammation. Important knowledge has been gained on the symptom dimensions that inflammation is driving and the mechanisms of action of cytokines in the brain, providing new targets for drug research and development. The aim of the book “Inflammation-Associated Depression” is to present this field of research and its implications in a didactic and comprehensive manner to basic and clinical scientists, psychiatrists, physicians, and students at the graduate level.
出版日期Book 2017
关键词Biological Psychiatry; Depression; Inflammation; Cytokine; Indoleamine 2,3 dioxygenase; Kynurenine; Phenyl
版次1
doihttps://doi.org/10.1007/978-3-319-51152-8
isbn_softcover978-3-319-84581-4
isbn_ebook978-3-319-51152-8Series ISSN 1866-3370 Series E-ISSN 1866-3389
issn_series 1866-3370
copyrightSpringer International Publishing AG 2017
The information of publication is updating

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