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Titlebook: Inflammation and Demyelination in the Central Nervous System; International Congre Byron H. Waksman (Director of Research Programms), Confe

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,Etiology and Pathogenesis of Monophasic and Relapsing Inflammatory Demyelination — Human and Experi variants of this disease have been extensively studied during the past decades and from the view of pathohistology can be regarded as suitable models for acute inflammatory demyelinating diseases in humans (acute perivenous leucoencephalomyelitis and acute hemorrhagic leucoencephalomyelitis; Alvord
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Virologic Models of Chronic Relapsing Demyelinating Disease,rain of mouse hepatitis virus (MHV), a coronavirus (3–6), the other caused by Theiler’s murine encephalomyelitis virus (TMEV), a picornavirus (7–10). Different pathogenic mechanisms are responsible for demyelination in these diseases. MHV-induced demyelination follows the lytic infection of oligoden
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Circulating Myelinotoxic Factors in Human and Experimental Demyelinative Disease,ot clearly explored yet. However, demyelinating agents have been suggested to be present in the circulating fluid, lymphocytes and brain tissue. The agent was first described by Bornstein and Appel (1) in 1961. They applied sera from EAE rabbits to the myelinated cerebellar cultures and found the se
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Neurochemical and Model Membrane Studies in Demyelinating Diseases,al evidence in the biomembrane field has shown that membrane structures completely different in their chemical composition, metabolism or permeability, are nevertheless indistinguishable from their ultrastructural morphology.
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Immunologic Models of Acute and Chronic Inflammation,athologic mechanisms (Boese, ed., 1980; Waksman, 1981). These were first distinguished over a half century ago, but the detailed cellular and molecular events underlying each are still in the process of elucidation.
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https://doi.org/10.1007/978-3-642-69094-5Entmarkungskrankheit; Entzündung; Inflammation; System; Zentralnervensystem /Erkrankung; antigen; infectio
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