Titlebook: Inflammation; Andrzej Górski,Hubert Krotkiewski,Michał Zimecki Book 2001 Springer Science+Business Media B.V. 2001 Asthma.Chemokine.antibi

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Interleukin 15: Its Role in Inflammation and Immunity,cell proliferative activity. It utilizes the signal-transducing β/γ polypeptides of the IL-2 receptor complex, thus sharing many biological activities with IL-2, in addition to its high-affinity private receptor subunit IL-15Rα. Accumulating evidence indicates that the biological relevance of IL-15

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Cytokines and Costimulatory Molecules: Positive and Negative Regulation of the Immune Response to ,nes and their effects have been described on a myriad of cellular functions. Cytokine interactions may not be linear, thus making the system extremely intricate and with unpredictable features. Therefore, each model of disease may be unique, with its own mechanism of autoregulation dictated by posit

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Nuclear Import of Arachidonate 5-Lipoxygenase,nase catalyzes the rate-limiting first two steps in the synthesis of leukotrienes from arachidonic acid. Although it has long been known that 5-lipoxygenase moves from the cytoplasm to a membrane . activation, it has only recently been recognized that the enzyme may shuttle into and out of the nucle

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Suppression of Mast Cell Activation by Glucocorticoid,en and IgE antibodies, release of chemical mediators is followed by secretion of multiple cytokines. We report that IL-3-dependent mucosal-type mast cells undergo apoptosis when IL-3 is withdrawn. In addition, cross-linking of high affinity IgE receptors prevents apoptosis of mast cells by paracrine

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Pathogenetic Mechanisms of Atopic Dermatitis,red lipid composition of the stratum corneum is responsible for the xerotic aspect of the skin and determines a higher permeability to allergens and irritants. Keratinocytes of AD patients exhibit a propensity to an exaggerated production of cytokines and chemokines, a phenomenon that can have a maj

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coagulation

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Mechanisms of , Pathogenesis,route and respiratory route. . is capable of invading mucosal epithelial cells and translocating across the mucosa. The bacterium can infect macrophages, interfering with several functions of the host cell. The host defense against . is primarily dependent on CD4. T lymphocytes and natural killer ce

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