Titlebook: Improving Prognosis for Kidney Disorders; Morrell Michael Avram Book 2002 Springer Science+Business Media Dordrecht 2002 blood pressure.ca

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Metabolic Pathogenesis of Cardio-Renal Disease,e in patients with diabetes and in those with essential hypertension. Moreover, changes in kidney function, even with modest as reductions in estimated creatinine clearance to approximately 60 ml/min/1.73m. or a serum creatinine of > 1.4, is associated with increased cardiovascular risk. Treatment w

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Oxidative Stress and Inflammation in Hemodialysis Patients,ps of factors have been identified as being responsible. These include the traditional causes such as hypertension, hyperlipidemia, diabetes, smoking and race, and factors arising from the underlying renal disease and the dialysis procedure itself. The relationship between these factors and the deve

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Prognostic Value of Enrollment Parathyroid Hormone in Hemodialysis and Peritoneal Dialysis Patientsthe relationship of enrollment serum intact PTH, as well as various demographic characteristics and other biochemical parameters, to all-cause mortality in hemodialysis (HD) and peritoneal dialysis (PD) patients. We enrolled 345 HD and 277 PD patients and monitored the patients up to 14 years. Obser

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Prealbumin As an Important Predictor for Survival and Nutritional Status in Hemodialysis and Peritooneal dialysis (PD) patients. We, and others, have previously reported that single enrollment levels of serum nutritional markers such as albumin, cholesterol, creatinine, and pre-albumin are directly correlated with mortality in HD and PD patients. We have prospectively examined the relationship of

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How to Attain Optimal Antiproteinuric Dose of Losartan in Non-Diabetic Patients with Nephrotic Rangfinding studies of antihypertensives have only been based on blood pressure so far. The present study aimed to find the optimal antiproteinuric dose of the angiotensin II antagonist losartan. An open-label dose-response study using subsequent six-week treatment periods was performed in ten non-diabe

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Hypoalbuminemia in the Patient with Renal Disease: Its Causes and Consequences,y lower in women (.).. The development of hypoalbuminemia, then, must be traced to the factors that can change the primary variables that regulate its level in health. Virtually all causes of hypoalbuminemia carry with them the potential to cause morbidity. Thus the cause of hypoalbuminemia must be

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Kidney Disease and Mechanisms that Cause Loss of Muscle Mass,tent increase in the rate of protein degradation or decrease in protein synthesis will result in substantial loss of muscle mass and increased morbidity and mortality. Activation of protein degradation in the ubiquitin-proteasome system is the mechanism contributing to loss of muscle mass in kidney

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Role of Plasma Homocysteine and Plasminogen Activator Inhibitor-1 in the Progression of Diabetic Ne varies substantially between patients, ranging from 2 to 20 ml/min/year. Thus, identification of predictors of progression in diabetic nephropathy is of importance. Plasma total homocysteine (tHcy) rises with urinary albumin rate in diabetes, and plasminogen activator inhibitor-1 (PAI-1) has been c

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Characterization of the PKD 1 Gene Product, Polycystin-1,kb mRNA that is predicted to result in a 462kDa membrane protein. The gene product, polycystin-1, has a large extracellular portion comprised of a novel combination of protein-protein interacting domains and is postulated to be a plasma membrane receptor involved in cell-cell/matrix interactions. Sl