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Titlebook: Immunology of the Connective Tissue Diseases; G. S. Panayi (ARC Professor of Rheumatology) Book 1994 Springer Science+Business Media Dordr

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Cytokines and Inflammatory Arthritis, resorption, induction of inflammatory prostanoids, and lymphocyte, monocyte and endothelial cell activation. Systemic effects include pyrexia, altered sleep patterns, anorexia, cachexia and induction of the acute phase proteins1,2.
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Adhesion Molecules involved in Leukocyte-Endothelial Cell Interactions,ssion of inflammatory reactions. The last decade has seen a growing awareness of the basic mechanisms underlying leukocyte emigration and, in particular, the importance of the adhesion molecules which facilitate interactions between leukocytes and vascular endothelial cells (EC). The considerable ef
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The Complement System and Connective Tissue Disease,es of complement include: promotion of the inflammatory response; opsonization of pathogens; opsonization and clearance of immune complexes; target cell lysis; and the development of antibody responses. These diverse activities, bridging the inflammatory and adaptive immune systems, are compatible w
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Animal Models of Systemic Lupus Erythematosus, analysed without appropriate animal models. These models may help to define aetiology, determine pathogenesis,and design innovative therapies. Indeed, systemic lupus erythematosus(SLE), as well as other autoimmune diseases, has animal models. Not surprisingly, they vary considerably in the ‘closene
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Retroviral Arthritis in Animals and Man,ich are generally thought not to cause disease. The lentiviruses, which include ovine maedi—visna virus (MVV), caprine arthritis—encephalitis virus (CAEV) and the human and simian immunodeficiency viruses (HIV and SIV), cause chronic inflammatory and degenerative disease of the lungs, brain, joints,
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