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Titlebook: Immunology of Gastrointestinal Disease; T. T. MacDonald Book 1992 Springer Science+Business Media Dordrecht 1992 HIV.bacteria.diseases.gas

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Host responses to , infection,t pathogenic role for bacteria in this condition.. In 1983 and early 1984, however, three separate groups almost simultaneously reported their observations relating to the association between a newly recognized spiral organism and chronic gastritis, and this has focused attention firmly on the role this agent may have in peptic ulcer disease..
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The cells and tissues of the gastrointestinal tract,vulnerable to infection, since only a single epithelial layer separates the gut lumen and the tissues. Large numbers of leukocytes are present throughout the gut as single cells in the lamina propria and epithelium, and as aggregates in lymphoid follicles. For the most part the antigenic stimulus fo
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Immunology of gastrointestinal food allergy in infancy and early childhood,to a variety of causes; for example, a congenital digestive enzyme defect such as sucrase-isomaltase deficiency, an acquired digestive enzyme defect such as lactase deficiency secondary to small intestinal mucosa damage, or the conditions may be immunological in origin.
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Intestinal graft-versus-host disease,nts (BMT), and intestinal damage is also an invariable feature of most animal models of GVH disease. The pattern of pathology in intestinal GVH disease also reproduces a variety of other, naturally occurring enteropathies. Thus, a knowledge of the pathophysiology of intestinal GVH disease is importa
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