Titlebook: Immunological Aspects of Liver Disease; Howard C. Thomas,Peter A. Miescher,Hans J. Mueller Book 1982 Springer-Verlag Berlin Heidelberg 198

obdurate

书目名称Immunological Aspects of Liver Disease影响因子(影响力)




书目名称Immunological Aspects of Liver Disease影响因子(影响力)学科排名




书目名称Immunological Aspects of Liver Disease网络公开度




书目名称Immunological Aspects of Liver Disease网络公开度学科排名




书目名称Immunological Aspects of Liver Disease被引频次




书目名称Immunological Aspects of Liver Disease被引频次学科排名




书目名称Immunological Aspects of Liver Disease年度引用




书目名称Immunological Aspects of Liver Disease年度引用学科排名




书目名称Immunological Aspects of Liver Disease读者反馈




书目名称Immunological Aspects of Liver Disease读者反馈学科排名

ineptitude

Book 1982to "self‘ components. Autoimmune disease occurs as a failure ofthis system either as a result of a primary defect in the regulatory apparatus (primary autoimmunization) or because of a change in the antigenicity of the tissues (secondary autoimmunization). Autoaggressive reactions are characterised

recession

Drug-Induced Chronic Hepatitis,. All individuals are susceptible, the lesion is reproducible in animals and there is a direct dose relationship. Even after severe hepatic necrosis, should the patient survive, agents producing a direct hepatotoxic reaction seldom lead to a chronic lesion.

enormous

一再困扰

Genetic Determinants of Autoimmune Chronic Active Hepatitis,lity antigens, of relevance to tissue transplantation, and associations between certain immunopathic diseases and HLA specificities [26, 31]. However, despite much experimental work and theoretical speculation, no unifying concept relating MHC-linked Ir genes effects and MHC- linked disease susceptibility has yet emerged.

HEAVY

Urea508

大量

Immunopathogenesis of the Extrahepatic Manifestations of Hepatitis B Virus Infection,ese syndromes and their comparison with animal models of immune complex injury, we have made progress in understanding the pathogenesis of these extrahepatic features; however, many important questions about the association among HBV, immune complexes, and extrahepatic tissue injury remain unanswered.

太空

繁殖

Pathogenic Mechanisms in Primary Biliary Cirrhosis,moral immunity. Patients exhibit a generalised state of immune hyporesponsiveness being anergic to DNCB [15] and producing a diminished antibody response to parenteral antigenic challenge [16, 57]. They also exhibit failure of the immune response to convert from IgM to IgG antibody synthesis [57].