Titlebook: Immune Mechanisms in Inflammatory Bowel Disease; Richard S. Blumberg,Markus F. Neurath Book 2006 The Editor(s) (if applicable) and The Aut

CILIA

书目名称Immune Mechanisms in Inflammatory Bowel Disease影响因子(影响力)




书目名称Immune Mechanisms in Inflammatory Bowel Disease影响因子(影响力)学科排名




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书目名称Immune Mechanisms in Inflammatory Bowel Disease网络公开度学科排名




书目名称Immune Mechanisms in Inflammatory Bowel Disease被引频次




书目名称Immune Mechanisms in Inflammatory Bowel Disease被引频次学科排名




书目名称Immune Mechanisms in Inflammatory Bowel Disease年度引用




书目名称Immune Mechanisms in Inflammatory Bowel Disease年度引用学科排名




书目名称Immune Mechanisms in Inflammatory Bowel Disease读者反馈




书目名称Immune Mechanisms in Inflammatory Bowel Disease读者反馈学科排名

Anticoagulants

Overview of Role of the Immune System in the Pathogenesis of Inflammatory Bowel Disease,led that CD and UC are fundamentally different diseases. The former has the molecular imprints of a Th1 dominant cell-mediated hypersensitivity response whereas the latter appears to involve antibody-mediated hypersensitivity.

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0065-2598 mmatory Bowel Disease. This book broadly treats the topics that lead to understanding of the pathogenesis of this disease in an organized, systematic approach. .978-1-4419-2157-4978-0-387-33778-4Series ISSN 0065-2598 Series E-ISSN 2214-8019

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Pandemic

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Microbial and Dietary Factors in the Pathogenesis of Chronic, Immune-Mediated Intestinal Inflammati

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Involvement of Dendritic Cells in the Pathogenesis of Inflammatory Bowel Disease,

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Role of Mast Cells and Eosinophils in Neuroimmune Interactions Regulating Mucosal Inflammation in I

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Alterations of T Lymphocytes in Inflammatory Bowel Diseases,rsa, and thus experimental colitis is not explained as an imbalance between Th1 and Th2 subsets. At present the data are compatible with the concept that excessive responses of either the Th1 or Th2 effector subsets are detrimental and can result in inflammatory bowel disease, and that this is preve