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Titlebook: Idiopathic Pulmonary Fibrosis; A Comprehensive Clin Keith C. Meyer,Steven D. Nathan Book 2019Latest edition Springer Nature Switzerland AG

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Daniela J. Lamas,David J. Ledererly along the lines of constructing multivariate models which are based on a suitably chosen specific physical model rather than on what would seem to be a more general approach—that of extending the characterizing properties of univariate exponential distributions. Characterization theorems—with occ
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Paolo Spagnolo,Elisabetta Cocconcelli,Vincent Cottinly along the lines of constructing multivariate models which are based on a suitably chosen specific physical model rather than on what would seem to be a more general approach—that of extending the characterizing properties of univariate exponential distributions. Characterization theorems—with occ
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Idiopathic Pulmonary Fibrosis: The Epidemiology and Natural History of Disease,less and often insidious manner.” However, recent epidemiologic studies have revealed the true burden of this disease on society and identified risk factors for the development of disease that may ultimately allow not only for disease prevention but also for further insight into the pathobiology of
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Imaging of Idiopathic Pulmonary Fibrosis,tial pneumonia (UIP) represents the imaging and histopathologic correlate of IPF. Although chest radiographs often show abnormalities in patients with UIP, the findings are typically nonspecific. High-resolution computed tomography (HRCT) allows for accurate characterization of many types of pulmona
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Pulmonary Function Tests in Idiopathic Pulmonary Fibrosis,lung compliance caused by tissue stiffness leads to a restrictive ventilatory defect, while ventilation and perfusion abnormalities are associated with a reduction of lung diffusion for carbon monoxide (DLco). The consequence of these volume and diffusion alterations is a progressive gas exchange im
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Mechanisms of Fibrosis in IPF,atrix, which stiffens the lung and leads to respiratory failure. The pathobiology of IPF is characterized by the presence of alveolar epithelial cell injury and apoptosis, accompanied by a progressive fibrosis. Thus, while inflammatory signaling may play a role in IPF, the previous paradigm of an .
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Genetics of Pulmonary Fibrosis, sporadic interstitial lung disease (ILD) and, especially, idiopathic pulmonary fibrosis (IPF). Thus, these genetic insights have begun to reveal the molecular basis of a disease that was previously thought to be unknown. A common variant within the promoter of the . gene is the most replicated sing
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