Titlebook: Hypoparathyroidism; A Clinical Casebook Natalie E. Cusano Book 2020 Springer Nature Switzerland AG 2020 Acute hypocalcemia.Autoimmune hypop

spinal-stenosis

Norbert Bach,Carsten Brehm,Thorsten Petrydisorders. After this, less common causes include iron or copper overload, tumor infiltration of the parathyroid glands, or radiation treatment. Rare patients have inherited or genetic forms of hypoparathyroidism, either presenting as isolated hypoparathyroidism or with syndromic features..Rare gene

制造

septicemia

https://doi.org/10.1007/978-3-8349-8610-8s of Hypoparathyroidism. A number of genetic mutations have been identified over the years. Idiopathic/isolated Hypoparathyroidism has been further divided into problems of PTH secretion, parathyroid gland development, and defect in the PTH gene itself. A case of nonsyndromic patient with features o

reflection

FLOAT

Konzeption Empirische Literaturwissenschaftat regular intervals, as asymptomatic renal impairment may occur. If creatinine levels start rising, examination should be performed for urinary tract obstruction which may be due to the development of renal stones.

停止偿付

https://doi.org/10.1007/978-3-662-54777-9woman with a history of bilateral cataract surgery noted to have basal ganglia calcifications on imaging and hypocalcemia. She was subsequently diagnosed with hypoparathyroidism. Symptoms of cataract include blurry or double vision, faded colors, halos around light, sensitivity to light, glare, and

不透明性

forbid

转向

,Glaubwürdigkeit: ein Forschungsüberblick,ng term results in higher bone mass than in age- and sex-matched controls. The reduced activity in the bones is demonstrated by bone turnover markers below or in the lower part of the reference range. Whether the high bone mass in the disease changes the risk of fractures is not certain. One could s

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