Titlebook: Hormones, Intrauterine Health and Programming; Jonathan R Seckl,Yves Christen Book 2014 Springer International Publishing Switzerland 2014

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Developmental Programming and the Placenta: Focusing in on Glucocorticoids,een attributed to direct effects of glucocorticoids on the fetus, prematurely shifting tissue development from a proliferative to a more functionally mature state. However, fetal growth is dependent on a complex interplay of maternal, placental, and fetal endocrine signals, and glucocorticoid-mediat

不容置疑

Glucocorticoids, Programming and the Transmission of Effects Across Generations,tabolic and neuroendocrine disorders in adulthood, a phenomenon termed “early life programming.” One of the major hypotheses advanced to explain early life programming is fetal glucocorticoid overexposure occurring as a consequence of maternal stress, exogenous administration or dysfunction of the p

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The Role of the Placenta in Fetal Programming,ansfer of nutrients between mother and fetus and by responding to changes in environmental stimuli that affect maternal and/or fetal physiology. Placental size and function are exquisitely sensitive to alterations in maternal nutrition, oxygen tension and exposure to glucocorticoids. These factors a

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伪证

Human Fetal Growth Disorders and Imprinting Anomalies,esidues of CpG dinucleotides in gene promoters, transposons and imprinting control regions (ICR). Genomic imprinting refers to the epigenetic marking of genes that results in monoallelic expression, depending on their parental origin. Several hormone genes involved in embryonic and fetal growth are

竖琴

Epigenetic Effects of Extreme Intrauterine Growth in Humans,adult diseases. The field of study of developmental origins of health and disease (DOHaD) is embracing the use of epigenomic assays to explore this potential mechanistic process. These DOHaD investigations represent part of the spectrum of epigenome-wide association studies (EWAS), a new field of in

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