Titlebook: Homotopy Methods in Topological Fixed and Periodic Points Theory; Jerzy Jezierski,Wacław Marzantowicz Book 2006 Springer Science+Business

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reasingly seen as best addressed by the new fields of psychology and psychiatry. Speech, physical and occupational therapy took shape as professional practices. The Child Guidance movement arose, using psychology to address family dysfunction and social unrest. There were experiments with special ed

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ue function after prenatal administration of B2AR agonists. These studies have also shown that the fetal B2AR normally does not desensitize, and that several tissues can retain a fetal pattern of signaling after prenatal B2AR overstimulation. This type of dysregulated signaling may also be responsib

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c imbalance can leave a metabolic footprint that can be followed analytically to gain mechanistic insights into the pathophysiology and pathogenesis of autism and thereby open new windows for therapeutic intervention. Escalating evidence suggests that many autistic children may be under chronic oxid

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bnormalities within the CNS. Recently, our laboratory demonstrated the presence of neuroglial and innate neuroimmune system activation in brain tissue and cerebrospinal fluid of patients with autism, findings that support the view that neuroimmune abnormalities occur in the brain of patients with au

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ary data from our laboratory that suggest that . promotes postnatal cerebellar development via cell cycle regulation and interactions with extracellular growth signals. After reviewing these data from different disciplines, it is our hope that the reader will better understand how abnormal cerebella

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formationis used as an essential method, and the assertions of theorems typically state the existence of ?xed or periodic points for every map of the whole homotopy class, we refer to them as homotopy methods o978-90-481-6998-6978-1-4020-3931-7

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Bayesian Population Modeling of Effective Connectivity.ce criteria. The significance of resulting connectivity statistics are evaluated by permutation-based approximations to the null distribution. The method is demonstrated on simulated data and on actual multi-subject neurological time-series.

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