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Titlebook: High Altitude and Man; John B. West,Sukhamay Lahiri Book 1984 American Physiological Society 1984 High Altitude.Tibet.adaptation.aldostero

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发表于 2025-3-21 18:50:58 | 显示全部楼层 |阅读模式
书目名称High Altitude and Man
编辑John B. West,Sukhamay Lahiri
视频videohttp://file.papertrans.cn/427/426195/426195.mp4
概述Leading authorities on high-altitude physiology contribute to this work.Focuses on man at extreme altitude, sleep and restoration.Also focuses on physiology of permanent residents of high altitude
丛书名称Clinical Physiology
图书封面Titlebook: High Altitude and Man;  John B. West,Sukhamay Lahiri Book 1984 American Physiological Society 1984 High Altitude.Tibet.adaptation.aldostero
描述Leading authorities on high-altitude physiology contribute to this work, which is divided into three sections: Man at Extreme Altitude; Sleep and Restoration at High Altitude; and Physiology of Permanent Residents of High Altitude. Based on a symposium on physiology at high altitude sponsored by the American Physiological Society, the volume includes several chapters on the achievements of the 1981 American Medical Research Expedition to Mt. Everest, where the first physiological measurements at altitudes above 8,000 meters were recorded. With growing interest in the study of human performance in these conditions, this text marks a lasting achievement in high-altitude physiology.
出版日期Book 1984
关键词High Altitude; Tibet; adaptation; aldosterone; blood; brain; breathing; cell; hypoxia; physiology; population;
版次1
doihttps://doi.org/10.1007/978-1-4614-7525-5
isbn_ebook978-1-4614-7525-5
copyrightAmerican Physiological Society 1984
The information of publication is updating

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Effects of Acclimatization on Sleep Hypoxemia at Altituden saturation during sleep in a cross-sectional study comparing groups of subjects who had been at altitude (5,360 m) for varying lengths of time and therefore had different degrees of acclimatization (5, 10). We wondered if any lack of improvement in arterial oxygen saturation with acclimatization h
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John B. West. Thrombophilia refers to congenital or acquired predispositions to thrombus formation. Here, we review the currently established hereditary and acquired thrombophilias. We will review genetic thrombophilic conditions such as factor V Leiden, the prothrombin gene 20210 mutation, protein C and S defi
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F. Duane Blumeh of which provide different information needed for an accurate diagnosis. Removal of heparin alone is not sufficient to halt the pathology of HIT. A potent intravenous thrombin inhibitor (argatroban, bivalirudin) is used to treat HIT thrombosis. Other non-heparin anticoagulants are used to manage p
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Robert M. Winslowsecurity, as well as the State Department’s desire to exercise hegemony among the peoples of Asia and Africa in their revolt against European colonialism. It also served the interests of policymakers who sought to roll back the modest achievements of the New Deal and block the further expansion of t
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