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Titlebook: Hepatitis C Virus I; Cellular and Molecul Tatsuo Miyamura,Stanley M. Lemon,Takaji Wakita Book 2016 Springer Japan 2016 HCV.Immunity.Liver.P

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书目名称Hepatitis C Virus I
副标题Cellular and Molecul
编辑Tatsuo Miyamura,Stanley M. Lemon,Takaji Wakita
视频video
概述Written by top researchers who have led this field since the discovery of Hepatitis C Virus.Provides a concise and current review of the state of this rapidly advancing field.Strong focus on fundament
图书封面Titlebook: Hepatitis C Virus I; Cellular and Molecul Tatsuo Miyamura,Stanley M. Lemon,Takaji Wakita Book 2016 Springer Japan 2016 HCV.Immunity.Liver.P
描述.This volume is composed of chapters that review important fundamental aspects of HCV biology and disease pathogenesis including, for example, the discovery and identification of the HCV genome, early virus-cell interactions including identification of various cellular receptors, HCV gene expression studied using the HCV replicon system, identification and characterization of HCV structural- and non-structural HCV proteins, HCV replication in cultured cells, and host factors involved in viral replication. This volume also contains chapters dealing with immunity to HCV infection and pathogenesis. This is particularly important in understanding hepatitis C because HCV infection alone is not cell lytic. Mechanisms underlying the persistent nature of HCV infection are also discussed in these chapters. Many of the authors published articles that were listed among the “top 10 papers” published in the 24 years since HCV was discovered in 1989. Their citations are above 1,000 (Web of Science). The authors describe the background and significance of their contributions to the field in the context of findings from other research groups..
出版日期Book 2016
关键词HCV; Immunity; Liver; Pathogen; Replication Strategy; Virology; hepatology
版次1
doihttps://doi.org/10.1007/978-4-431-56098-2
isbn_softcover978-4-431-56775-2
isbn_ebook978-4-431-56098-2
copyrightSpringer Japan 2016
The information of publication is updating

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B Cell Responses and Control of HCV Infectionralization that are not associated with viral escape or escape with compromised fitness. The focus of this review is on the immunogenic determinants on E2, which are roughly segregated into the hypervariable region 1 (HVR1), and five clusters of overlapping epitopes, designated as antigenic domains
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Takaji Wakita M.D., Ph.D.g der Extremitätennerven entsprechen. Die den vorderen Extremitäten zugehörige kraniale Anschwellung liegt vorwiegend im Bereiche des Halsabschnitts und wird daher Intumescentia cervicalis (Abb. S. 31) genannt. Sie hat ihre größte Mächtigkeit in Höhe des 5. und 6. Halsnerven.
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