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Molecular Mechanisms of Lipotoxicity in Nonalcoholic Fatty Liver Disease,), the more severe form of NAFLD, is associated with increased risk of cardiovascular morbidity and mortality, insulin resistance (IR), type 2 diabetes mellitus (T2DM), hepatic steatosis, elevated circulating levels of free fatty acids (FFAs) and hepatocyte apoptosis. Because lipid-induced toxicity,

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Lipid Droplet Proteins and Hepatic Lipid Metabolism, passive depot of excess lipids but is a metabolically active organelle that interacts with endoplasmic reticulum, mitochondria and other cellular compartment to regulate fate of intracellular lipids. As the increase in size and number of LD is a key feature of hepatosteatosis, information regarding

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Influence of Gut Microbiota on Hepatic Lipogenesis and Disease Pathogenesis,y of biochemical pathways that are central in keeping energy homeostasis. Loss of this delicate balance, however, may lead to a spectrum of liver diseases such as NAFLD, NASH, fibrosis, cirrhosis and HCC. The etiology of liver disorders is often multifactorial and greatly influenced by host genetics

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