Titlebook: Handbook of Diagnostic Endocrinology; Janet E. Hall,Lynnette K. Nieman Book 2003 Springer Science+Business Media New York 2003 Diabetes.Go

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Hyper- and Hypocalcemia,m is found in bone, with the remaining fraction in either the extracellular or intracellular compartments of all other tissues. Approximately half of the circulating blood calcium is bound to serum proteins, but it is the nonbound fraction (or ionized calcium) that is tightly controlled by the calci

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Menstrual Dysfunction,ypothalamus, the pituitary, and the ovary. This classic endocrine cascade is initiated by pulsatile secretion of gonadotropin-releasing hormone (GnRH) from the hypothalamus into the pituitary portal venous system. The subsequent release of follicle-stimulating hormone (FSH) and luteinizing hormone (

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Differential Diagnosis and Evaluation of Hyperandrogenism,emale population. The most common cause of hyperandrogenism is the polycystic ovary syndrome (PCOS), with nonclassic adrenal hyperplasia (NCAH), androgen-secreting tumors, and androgenic drug intake being much less frequent. Hyperandrogenism, the endocrine disorder, should be distinguished from derm

Cerebrovascular

Investitions- und Finanzplanung,tion .,. The majority of hormonal proteins and steroids are bioactive at extremely low concentrations in the peripheral circulation, and hormone metabolites often play a significant physiological role at target tissue sites. Therefore, evaluation of a patient’s endocrine status with a given immunoas

minion

https://doi.org/10.1007/978-3-322-98983-3ferent disease states can potentially disrupt the finely balanced mechanisms that control the intake and output of water and solute. Since body water is the primary determinant of the osmolality of the extracellular fluid (ECF), disorders of water metabolism can be broadly divided into hypoosmolar d

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macular-edema