Titlebook: Haemostatic Failure in Liver Disease; P. Fondu,O. Thijs Book 1984 Martinus Nijhoff Publishers, Boston 1984 blood.diseases.liver.liver dise

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Coagulation factors synthesis by the liver, with special reference to Factor VIII and Factor V,teins, including coagulation factors. Many studies, utilising a variety of techniques, some of which are listed briefly in Table 1, have provided fundamental information for the diagnosis and treatment of hepatic disease.

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Oral Anticoagulants: Unexpected Side-Effects or New Fields of Application?,l. It is shown, that the dosages of warfarin, required for a partial inhibition of the clotting factor synthesis also affects the non-hepatic vitamin K-dependent systems. The presence of non-hepatic vitamin K-dependent carboxylase is demonstrated in various rat and bovine tissues as well as in human

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Clinical Manifestations of the Haemostatic Failure in Acute and Chronic Liver Disease,c varices and erosions. Haemorrhags from frank peptic ulceration can also occur but is much less frequent. In the terminal stages of liver failure generalised bleeding into the skin and mucous membranes is seen. Retroperitoneal haemorrhage, bleeding from the nasopharynx, bronchial free, uterus and b

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Coagulation Defects Following Peritoneovenous Shunts,elated to diffuse bleeding in these patients has hindered the use of this procedure in such indications (8). The results of the large series recently published and of coagulation studies, have contributed to a better knowledge of the prevalence of coagulation abnormalities after peritoneovenous shun

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AT-III Concentrate Treatment: Current Experience,embolic episodes already at young age (2). The acquired deficiency state as for example occurring in liver cirrhosis is a much more frequently observed condition. Antithrombin-III (AT-III) is synthesized in the liver and its synthesis nas also been shown to occur m cultured endothelial cells (3). De

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Auteur Meets Genre: Rohmer and the Rom-Com C, has anticoagulant properties. The rate of . activation of purified Protein C by thrombin is too slow to have any physiological importance. However, infusion of thrombin in dogs leads to the formation of anticoagulant activity in the circulation within 5 min after starting the infusion (3). The t