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Titlebook: Free Radicals and Inflammation; Paul G. Winyard,David R. Blake,Christopher H. Evan Book 2000 Springer Basel AG 2000 Arthritis.apoptosis.bi

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书目名称Free Radicals and Inflammation
编辑Paul G. Winyard,David R. Blake,Christopher H. Evan
视频video
丛书名称Progress in Inflammation Research
图书封面Titlebook: Free Radicals and Inflammation;  Paul G. Winyard,David R. Blake,Christopher H. Evan Book 2000 Springer Basel AG 2000 Arthritis.apoptosis.bi
描述As in so many fields of scientific endeavour following the molecular biology revo­ lution, our knowledge of the role of radicals not only in pathological states, but in basic physiology has developed exponentially. Indeed, our evolving concepts have, like so many political parties, been forced into dramatic "V-turns" and contortions. Within our working lives, we have had to debate whether radicals made any con­ tribution to any pathology, whilst now it is difficult not to entertain the view that every physiological process is pivotally controlled by exquisitely sensitive radical reactions. Inflammation is, of course, an example of pathology evolving from physiology, and in this book we have called upon both scientists and clinicians who have research interests in the complex switching mechanisms that sustain these transi­ tions. The book as a whole explores, from a physiological standpoint, how deter­ ministic radical systems sensitive to their initial conditions can interdigitate, iterate and feed back to control diverse cellular processes that create the inflammatory response. Whilst systems such as these to a mathematician would provide the basis for a chaotic response, one is f
出版日期Book 2000
关键词Arthritis; apoptosis; biology; cell; cell death; diseases; inflammation; nitrogen; rheumatism; rheumatoid art
版次1
doihttps://doi.org/10.1007/978-3-0348-8482-2
isbn_softcover978-3-0348-9586-6
isbn_ebook978-3-0348-8482-2Series ISSN 1422-7746 Series E-ISSN 2296-4525
issn_series 1422-7746
copyrightSpringer Basel AG 2000
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