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Titlebook: Experimental Myopathies and Muscular Dystrophy; Studies in the Forma Rainer Heene Book 1975 Springer-Verlag Berlin · Heidelberg 1975 histol

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发表于 2025-3-21 19:05:50 | 显示全部楼层 |阅读模式
书目名称Experimental Myopathies and Muscular Dystrophy
副标题Studies in the Forma
编辑Rainer Heene
视频video
丛书名称Schriftenreihe Neurologie‘ Neurology Series
图书封面Titlebook: Experimental Myopathies and Muscular Dystrophy; Studies in the Forma Rainer Heene Book 1975 Springer-Verlag Berlin · Heidelberg 1975 histol
出版日期Book 1975
关键词histology; liver; muscle; muscular dystrophy; pathogenesis; skeletal muscle; treatment
版次1
doihttps://doi.org/10.1007/978-3-642-66200-3
isbn_softcover978-3-642-66202-7
isbn_ebook978-3-642-66200-3Series ISSN 0080-715X
issn_series 0080-715X
copyrightSpringer-Verlag Berlin · Heidelberg 1975
The information of publication is updating

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Experimental Myopathies,nied by varying degrees of eosinophilia of the sarcoplasm. Since histochemical techniques were not used, predilective involvement of one or the other type of muscle fibres could not be demonstrated. Even under prolonged treatment, when severe changes were present in skeletal muscle, the myocardium a
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The Myopathy of 2,4-Dichlorophenoxyacetic Acid (2,4-D) in the Rat,fibres in an experimental disease. By appropriate dosage of this agent, acute or subacute myopathic states can be evoked in warm-blooded animals. The principal subject of the present study is the subacute myopathy of 2,4-D induced in the white rat by repeated injections of the substance. Preceding t
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Results,ns will be inhibited if the aqueous incubation is performed in the presence of 2,4-D at a concentration of 14 mM (S. in Table 3). The same applies to the Phosphorylase reaction in the gelatan films into which 2,4-D has been incorporated (Gg). In striking contrast to this, no apparent decrease ofPhos
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Matthew A. Glaser,Noel A. Clarkl interest. Among these, the hereditary myopathies of the mouse and of the chicken and the myopathy induced by vitamin-E deficiency are particularly important, as well as the myopathies that follow treatment with corticosteroids or chloroquine. Morphological and physiological studies of these model
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Topological Hypothesis on the Origin,nied by varying degrees of eosinophilia of the sarcoplasm. Since histochemical techniques were not used, predilective involvement of one or the other type of muscle fibres could not be demonstrated. Even under prolonged treatment, when severe changes were present in skeletal muscle, the myocardium a
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