Titlebook: Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond; Bernward Garthoff,Andreas M. Knorr,Friedel Seuter Conference

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https://doi.org/10.1007/978-3-642-79338-7Calcium antagonists; Calciumantagonisten; Hochdruck; Pharmakologie; Stroke; hypertension; pathophysiology;

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https://doi.org/10.1007/978-3-030-75875-2Inhibition of atrial natriuretic peptide (ANP) degradation by enkephalinase is known to increase endogenous ANP levels and to induce similar functional effects as administration of exogenous ANP. The influence of this principle on cardiac function in rats with hypertension-induced heart failure was investigated.

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Sinorphan Improves Cardiac Structure and Function in Aged Stroke-Prone Spontaneously Hypertensive RInhibition of atrial natriuretic peptide (ANP) degradation by enkephalinase is known to increase endogenous ANP levels and to induce similar functional effects as administration of exogenous ANP. The influence of this principle on cardiac function in rats with hypertension-induced heart failure was investigated.

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,Die Genomgröße bei Pflanzen ist dynamisch,oth muscle cells (SMC) in carotid arteries of rabbits which were exposed to a local atherogenous stimulus. These substances have been recommended as antiatherogenic drugs. However, when using animal models for studying drug effects on the development of intimal proliferation of vascular SMC in the c

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Erich Grießler,Harald Rohracheron et al. 1987; Boonen and De Mey 1990; Bodin et al. 1993). This hyperre-activity has been suggested to arise either from a change in receptor affinity (Nyborg and Bevan 1988), in receptor number, or in the transduction mechanisms of the receptor (Asano et al. 1988; Johnson et al. 1991). In addition

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