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Titlebook: Experimental Hematology Today—1989; Selected Papers from Norbert C. Gorin,Luc Douay Conference proceedings 1990 Springer-Verlag New York In

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Effects of Ubenimex on Proliferation and Differentiation of Human Bone Marrow Cells and Leukemic CelO), granulocyte-macrophage colony-stimulating factor (GM-CSF) and granulocyte colony-stimulating factor (G-CSF). Ubenimex significantly enhanced G- and GM-CSF- induced colony formations (derived from CFU-G and CFU-GM, respectively) of human bone marrow cells at concentrations of 0.001, 0.01, 0.1 and
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Stimulation of the Immune System for the Therapy of Advanced Stage Neuroblastomad autologous bone marrow transplantation (ABMT) (1). However, relapses occuring up to 72 months post-graft are of major concern; spontaneous regression of this tumor in children below 1 year of age favors a role of the immune system in antitumoral defense and the potential benefit of immunotherapy.
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Coping Strategies and Genocide Preventionportunities for new approaches to cancer therapy. The preferential retention of intravenously administered hematoporphyrin derivative (HPD) by tumor tissue is now exploited with encouraging results in the treatment of head and neck, lung, breast and bladder cancer, and several other solid tumors [1, 2].
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K. P. Gopinathan,Omana Joy,Amit Singhrane receptors for interleukin 2 (IL2R) [2]. The expression of these receptors on T cells was shown to be a transient event [3] and restimulation with either antigen or mitogen is required for continous IL2R expression and maintainance of T cell proliferative capacity.
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Genome Editing and Biological Weapons, in our experience, while leukemic blast cell recovery and viability are well preserved after thawing, an unusual rate of defective leukemic colony (AML-CFU) recovery has been observed when using freezing techniques which are usually efficient for normal bone marrow progenitors.
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Phage Integrases for Genome Editing, have significantly reduced collagen I mRNA expression when compared to cloned stromal cell lines from which they were derived. Similar patterns of growth factor and collagen I mRNA expression in stromal cells are induced by inflammatory mediators IL-1 and TNF.
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Marrow Stromal Cells, Hematopoietic Growth Factors and Extracellular Matrix Proteins have significantly reduced collagen I mRNA expression when compared to cloned stromal cell lines from which they were derived. Similar patterns of growth factor and collagen I mRNA expression in stromal cells are induced by inflammatory mediators IL-1 and TNF.
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