Titlebook: Exon Skipping and Inclusion Therapies; Methods and Protocol Toshifumi Yokota,Rika Maruyama Book 2018 Springer Science+Business Media, LLC,

短暂

书目名称Exon Skipping and Inclusion Therapies影响因子(影响力)




书目名称Exon Skipping and Inclusion Therapies影响因子(影响力)学科排名




书目名称Exon Skipping and Inclusion Therapies网络公开度




书目名称Exon Skipping and Inclusion Therapies网络公开度学科排名




书目名称Exon Skipping and Inclusion Therapies被引频次




书目名称Exon Skipping and Inclusion Therapies被引频次学科排名




书目名称Exon Skipping and Inclusion Therapies年度引用




书目名称Exon Skipping and Inclusion Therapies年度引用学科排名




书目名称Exon Skipping and Inclusion Therapies读者反馈




书目名称Exon Skipping and Inclusion Therapies读者反馈学科排名

COWER

恃强凌弱

短程旅游

https://doi.org/10.1007/978-3-031-14406-6ase. Defects in RNA splicing account for at least 10% of all genetic disorders, with the number expected to increase as more information is uncovered on the contribution of noncoding genomic regions to disease. Splice modulation through the use of antisense oligonucleotides (AOs) has emerged as a pr

MUT

Giordano Zambelli,Luciano Morgantiproach uses antisense oligonucleotides (AON) to modify the splicing of pre-mRNA to correct the mutation responsible for a disease, or to suppress a particular gene expression, as in allergic diseases. Antisense-mediated exon skipping is most extensively studied in Duchenne muscular dystrophy (DMD) a

multiply

Ultrasound, Embodiment and Abortion,tein. Insufficient levels of SMN results in the loss of motor neurons, which causes muscle weakness, respiratory distress, and paralysis. A nearly identical gene (.) contains a C-to-T transition which excludes exon 7 from 90% of the mature mRNA transcripts, leading to unstable proteins which are tar

multiply

mercenary

创新

Critical