Titlebook: Excitatory Amino Acids and Epilepsy; Robert Schwarcz,Yehezkel Ben-Ari Book 1986 The Editor(s) (if applicable) and The Author(s), under exc

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,Formspulenwickelung für Wechselstrom von ,,1983). Although the mechanisms of this permeability change are not completely understood, free arachidonic acid (Bazan, 1970; Rodriguez de Turco et al., 1983; Siesjö et al., 1983) has been implicated (Chan et al., 1983). Several other characteristics of the change are known. The morphologic basis fo

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Endogenous Excitotoxins as Possible Mediators of Ischemic and Hypoglycemic Brain Damageetween these three conditions (Siesjö and Wieloch, 1986), suggesting that at least in some aspects different pathophysiological mechanisms prevail. Although this selective neuronal damage has been known for several decades (Spielmeyer, 1925), its pathophysiological and molecular mechanisms are largely elusive.

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https://doi.org/10.1007/978-3-658-13294-1pathologies (Sanders, 1874; Hamilton, 1882; Anderson, 1887). Hence, the Jacksonian concept of hyperexcitable tissue adjacent to neocortical damage was accepted; however, that concept was not extended to the mesial temporal lobe (archicortex), where damage was well -established in epileptics.

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Metabolic, Morphologic and Electrophysiologic Profiles of Human Temporal Lobe Foci: An Attempt at Copathologies (Sanders, 1874; Hamilton, 1882; Anderson, 1887). Hence, the Jacksonian concept of hyperexcitable tissue adjacent to neocortical damage was accepted; however, that concept was not extended to the mesial temporal lobe (archicortex), where damage was well -established in epileptics.