Titlebook: Epstein-Barr Virus and Human Disease • 1988; Dharam V. Ablashi,Alberto Faggioni,Gary R. Pearson Book 1989 The Humana Press Inc. 1989 DNA.P

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Karoliina A. Koho,Elisa Piña-Ochoae human body via the oral route and shows primary replication predominantly in the tonsils. From there the virus is spread throughout the body via lymphoid cells. We have to assume that a widespread spectrum of cells, including the epithelial lineage becomes latently infected. The immune system cont

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https://doi.org/10.1007/0-306-47954-0pecific regulatory factors that may interact by binding to the promoter regulatory sequences have lagged in EBV systems. Here we summarize the main points of new studies that are the first to detect proteins binding to a key EBV promoter. We selected for our initial studies the presumptive promoter

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Methodischer Aufbau der Untersuchung,ls with phorbol ester, which activates the EBV replicative life cycle, induced the production of two cellular competitors which upon competition uncoupled the binding of EBNA-1 to ori-P. Gel retardation assay, DNase I footprinting and methylation interference analysis showed that the two cellular co

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978-1-4612-8852-7The Humana Press Inc. 1989

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Zielsetzung, Fragestellungen und Hypothesen,). The duplicated left (DL) promoter is almost a perfect copy of the DR promoter region (Figure 1F) (1) and directs the expression of early mRNAs containing short tandem repeats, the IR2 repeats which are partially homologous to the IR4 repeats (4, 5, 6).