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Titlebook: Epithelial Secretion of Water and Electrolytes; J. A. Young,P. Y. D. Wong Book 1990 Springer-Verlag Berlin Heidelberg 1990 Ionenkanäle.Muk

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书目名称Epithelial Secretion of Water and Electrolytes
编辑J. A. Young,P. Y. D. Wong
视频video
图书封面Titlebook: Epithelial Secretion of Water and Electrolytes;  J. A. Young,P. Y. D. Wong Book 1990 Springer-Verlag Berlin Heidelberg 1990 Ionenkanäle.Muk
出版日期Book 1990
关键词Ionenkanäle; Mukoviszidose; Pancreas; Sekretion; epithelialer Transport; exokrine Drüsen; fat; liver; physio
版次1
doihttps://doi.org/10.1007/978-3-642-75033-5
isbn_softcover978-3-642-75035-9
isbn_ebook978-3-642-75033-5
copyrightSpringer-Verlag Berlin Heidelberg 1990
The information of publication is updating

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Anhang zu Konstruieren und Berechnennds (see for a review, Petersen 1980). In the pancreas, all the cells from the same acinus are electrically coupled and in some cases, cells from different acini seem also to be coupled (Iwatsuki and Petersen 1978 a).
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Bruno Jacson Martynhak,Roberto Andreatinirved in preparations of isolated rough endoplasmic reticulum (RER) from the rat exocrine pancreas that the active reuptake of Ca. ions is associated with an anion conductance (Bayerdörffer et al. 1984; Kemmer et al. 1987) and it can be assumed that this permeability to anions serves to maintain elec
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Epithelial Chloride Channelsreting epithelia that exist, it may turn out that the models described above may only be extremes and that intermediate constellations may also occur (Marty et al. 1984; Young et al. 1988). The currently known mechanisms of channel activation make it seem likely that we will have to deal with more t
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Cation Channels and Secretiontributed among the secretory epithelia. The present review will first deal with the possible roles of these and other cation-selective channels in epithelial secretion and then with their distribution and characteristics.
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Theorie der Bildung des Anilinschwarz,d reason, which is not unconnected with the first, is that a number of identifiable disease states appear to be associated with a mutation affecting cellular transport mechanisms. The obvious examples are cystic fibrosis and chloridorrhoea.
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