Titlebook: Epicardial Adipose Tissue; From Cell to Clinic Gianluca Iacobellis Book 2020 Springer Nature Switzerland AG 2020 Cardiovascular risk and ep

抱狗不敢前

Heart Failure and Epicardial Adipose Tissue,le, although results are not univocal. Experimental studies suggest a potential protective role of the brown fat features of EAT against the systolic heart failure. A function of EAT p53 and adiponectin has been also suggested in patients with heart failure. A neuromodulatory role of EAT in heart fa

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Adrenal Secretome and Epicardial Adipose Tissue,aldosterone can lead to accumulation of epicardial adipose tissue (EAT), interfering with functional state. In fact, increased mineralocorticoid signaling is critically involved in the secretion of proinflammatory cytokines by EAT with consequent profibrotic cascade activation in the myocardium. In

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Targeting the Epicardial Adipose Tissue,icardial fat can serve as a target for pharmaceutical agents targeting the adipose tissue. EAT has shown to significantly respond to thiazolidinediones (TZD), glucagon-like peptide-1 receptor (GLP-1R) agonists, sodium-glucose cotransporter 2 inhibitors (SGLT2i), dipeptidyl peptidase-4 inhibitors (DP

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名义上

Pathology and Cardiotoxicity of the Epicardial Adipose Tissue,um, EAT can affect the morphology and function of all of the heart chambers. Increased epicardial fat has been largely associated with increased left ventricular mass, abnormal geometry, enlarged atria, and diastolic dysfunction. Multifactorial physical and biomolecular mechanisms can explain the effects of excessive EAT on the heart.

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Cardiometabolic Risk and Epicardial Adipose Tissue,Imaging of EAT can stratify the cardiovascular risk in subjects with type 2 diabetes and pre-diabetes. EAT increases the risk of cardiovascular diseases in patients with HIV, psoriasis, and postmenopausal women. EAT measurement can serve as a diagnostic tool in assessing cardiovascular and metabolic risk.

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