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Titlebook: Endometrial Cancer; Earl A. Surwit,David S. Alberts Book 1989 Kluwer Academic Publishers 1989 Staging.cancer.carcinoma.chemotherapy.endome

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https://doi.org/10.1007/978-1-4613-0867-6Staging; cancer; carcinoma; chemotherapy; endometrium; oncology; radiation therapy; surgical oncology
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Endometrial Cancer978-1-4613-0867-6Series ISSN 0927-3042 Series E-ISSN 2509-8497
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https://doi.org/10.1007/978-3-8350-5550-6y American women. A sizable body of evidence supports the role of both excessive endogenous estrogen and exogenous estrogen in the etiology of endometrial cancer. There is growing evidence that adequate progesterone has the opposite effect and that progestogen supplementation of postmenopausal estro
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Risking Antimicrobial Resistancepass the full extent of the malignant process. Such a process, when applied to individual patients, must take into account the extent of clinically detectable disease, the problems of spread, and the presumed preexisting clinically occult disease. Only then can adequate therapy be offered.
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https://doi.org/10.1007/978-3-642-18857-2 of the century [1], the clinical importance of malignant cells present in ascites or peritoneal washings was not addressed until 1956, when Keettel and Elkins [2] reported their preliminary experience with peritoneal cytology in ovarian cancer. In 1961, Morton et al. [3] demonstrated malignant cell
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Steven Ratuva,Hamdy A. Hassan,Radomir Compelary surgical and radiotherapeutic techniques. Although selected series have achieved somewhat better results, five-year survival rates worldwide are 75% for Stage I and 58% for Stage II endometrial carcinomas and have not substantially improved over the past few decades, despite high rates of operab
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ar [1]. Although surgery and radiation therapy are quite effective and achieve 80–90% cure rates in early stages, most of the fatalities occur in patients with advanced, recurrent, or metastatic disease.
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