Titlebook: Endocrinology of the Vasculature; James R. Sowers Book 1996 Humana Press Inc. 1996 Diabetes.Diabetes mellitus.growth factor.insulin.signal

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John T. Flynn,William S. Tasmann the vascular actions of insulin is also explored. The latter has important clinical consequences given that insulin resistance is a common feature of obesity, of noninsulin-dependent diabetes mellitus (NIDDM), and hypertension, and is associated with an increased risk of macrovascular disease (1,2)

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The Withdrawal and the End of the Cold War,ulates endothelial cell [Ca.]. (receptor mediated), promotes the release of prostacyclin (PGI), and facilitates inactivation of endothelial-derived relaxing factor (EDRF) or nitric oxide (NO) .,. LDL also stimulates [Ca.]. in vascular smooth muscle cells (VSMC) ., and in cardiac myocytes ..References

Mettle

Study Design/Method/Procedures,mboxane A2, which are vasoconstrictors as well as agonists of platelet aggregation. The endothelium also exerts a profound influence on vasomotion by its ability to metabolize circulating vasoactive substances such as serotonin, epinephrine, bradykinin, and angiotensin I. The endothelium elaborates

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Sidelined: The Millions Who Are Left Out,nd neuropathy in type I diabetic patients. However, the evidence that good glycemic control can prevent or slow the progression ofmacrovascular diseases is not clear . In this chapter, we discuss the cellular and molecular mechanisms that may explain the underlying pathological and clinical abnormal

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Retroperitoneoscopy in Children, emerging. This chapter reviews effects of sex hormones on the vasculature with emphasis on the processes that influence the progression of atherosclerosis and thus alter cardiovascular risk. Effects of postmenopausal hormone replacement on cardiovascular risk are reviewed.

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Endothelium-Derived Relaxing Factormboxane A2, which are vasoconstrictors as well as agonists of platelet aggregation. The endothelium also exerts a profound influence on vasomotion by its ability to metabolize circulating vasoactive substances such as serotonin, epinephrine, bradykinin, and angiotensin I. The endothelium elaborates

Gratuitous

Vascular Abnormalities in Diabetes Mellitusnd neuropathy in type I diabetic patients. However, the evidence that good glycemic control can prevent or slow the progression ofmacrovascular diseases is not clear . In this chapter, we discuss the cellular and molecular mechanisms that may explain the underlying pathological and clinical abnormal

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Sex Hormones and the Vasculature emerging. This chapter reviews effects of sex hormones on the vasculature with emphasis on the processes that influence the progression of atherosclerosis and thus alter cardiovascular risk. Effects of postmenopausal hormone replacement on cardiovascular risk are reviewed.

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