Titlebook: Emergence and Control of Zoonotic Viral Encephalitides; Charles H. Calisher,Diane E. Griffin Book 2004 Springer-Verlag Wien 2004 CNS.West

青石板

flaunt

Subversive neuroinvasive strategy of rabies virus,mal. It enters the nervous system via a motor neuron through the neuromuscular junction, or via a sensory nerve through nerve spindles. It then travels from one neuron to the next, along the spinal cord to the brain and the salivary glands. The virions are then excreted in the saliva of the animal a

绝种

Bridle

Charitable

Semliki Forest virus infection of laboratory mice: a model to study the pathogenesis of viral encepg extra neural inoculation the virus is efficiently neuroinvasive and crosses the blood-brain barrier to initiate perivascular foci of infection in neurons and oligodendrocytes. The outcome of infection ranges from clinically unapparent mild encephalitis to fatal panencephalitis. SFV infections of t

CHAFE

GRAVE

Tick-borne encephalitis and the impact of vaccination, disease in humans each year. The virus is primarily transmitted to humans when infected ticks take a blood meal, but infections due to the consumption of unpasteurized milk, primarily from goats, occur in certain regions. Based on genetic analyses, three closely related subtypes can be distinguishe

Graduated

archaeology

adduction

Molecular determinants of virulence of West Nile virus in North America,ve in these laboratory rodents. Other strains, such as Ethiopia 76a from lineage I, are not neuroinvasive and represent important viruses which can be used to elucidate the molecular basis of virulence and attenuation of WNY. To identify putative molecular determinants of virulence and attenuation,