Titlebook: Electrical Stimulation and Neuromuscular Disorders; W. A. Nix,G. Vrbová Conference proceedings 1986 Springer-Verlag Berlin Heidelberg 1986

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Stimulation of Denervated Muscle: What Do Isometric and Isotonic Recordings Tell Us? the arm). Only if a constant speed of movement is reached, will the shortening be truly isotonic. As the movement is ended and the arm is slowed, the inertia of the mass will exert a force in the opposite direction, which has to be opposed by action of antagonists. Finally, isometric contraction wi

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Effect of Electrical Stimulation on Denervated Musclee beneficial effect of this procedure in patients. Along with the clinical trials animal experiments have been conducted, yet many of these are impossible to compare, because the conditions of the experiments, such as stimuli parameters, duration of treatment, and conditions under which the muscle c

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Conference proceedings 1986ing of nerve-muscle interaction has shown that certain activity patterns can influence muscle. These experi­ mental results provide a rational basis for a possible therapeutic use of electrical stimulation of nerve and muscle. Previously most research has been conducted in normal tissue, and little

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Effects of Electrical Stimulation on Normal and Diseased Human Muscleich ultimately result in complete transformation to slow contracting fibres (Salmons and Vrbová 1969). There is overwhelming evidence that these changes are directly related to imposed activity and that functional and metabolic characteristics of muscle fibres depend upon the activity of the motoneuron.

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Conference proceedings 1986 a position of conflict between his desire to help and his awareness that there is no treatment. In this situation it is tempting to indiscriminately use any procedure that avoids an admission of medical helplessness while satisfying the patient‘s demand for treatment. Electrical interventions are o

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Das sog. Schleudertrauma der Halswirbelsäulerolled experimental conditions. This paper is concerned primarily with the reaction of the parent neuron to damage and the relation of functional changes in the neuron to repair and regeneration of the lost axon and to alterations in the level of neural activity.