Titlebook: Early Nutrition Programming and Health Outcomes in Later Life: Obesity and beyond; Berthold Koletzko,Tamás Decsi,Anne Hunty Conference pro

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https://doi.org/10.1007/978-3-642-54398-2global reduction in oxidative phosphorylation and electron transport chain genes and this translates into mitochondrial dysfunction in selected tissues. PGC1βKO mice also demonstrate blunted responses to physiological stresses such as cold exposure in BAT, adrenergic stimulation in BAT and heart and

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Security Assessment and Testing,ectively). Associations were independent of gender, parity, infant diets, and, for weight gain, birth weight SDS. Conclusion: Higher birth weight was associated with higher fat mass at 3 months whereas rapid weight gain in the first 3 months was associated with both fat and lean mass. Our data do no

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Infant Feeding and Later Obesity Risk,rd infant formulae (the “Early Protein Hypothesis”). These hypotheses are tested in the European Childhood Obesity Project, a randomized double blind intervention trial in more than 1,000 infants in five European countries (Belgium, Germany, Italy, Poland, Spain). Formula fed infants were randomized

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The Early Origins of Later Obesity: Pathways and Mechanisms,egulate energy balance. The difficulty in treating obesity, however, highlights the deficits in our current understanding of the pathophysiology which underlies the initiation and chronic nature of this disorder. Large population based studies in Europe and North America in healthy women and in wome

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Developmental Origins of Obesity: Programming of Food Intake or Physical Activity?,ably rendering them more susceptible to greater adipose tissue deposition. The central role that lean body mass has upon the ‘gating’ of energy sensing and the importance of regular physical activity for its potential to reduce the burden of a ‘thrifty phenotype’ will be briefly discussed in the pre