Titlebook: Dyslipidemias; Pathophysiology, Eva Abhimanyu Garg Book 2015 Humana Press 2015 Abetalipoproteinemia.Chronic kidney disease.Drug-induced dys

FOLLY

Julia A. Hubbard,Bing Xiao,Jon R. Wilsongher levels generally confer greater risk for cardiovascular disease (CVD) events. The key determinants for higher levels of atherogenic lipids measured as low-density lipoprotein cholesterol (LDL-C) include high dietary saturated fat, high dietary cholesterol, and low dietary polyunsaturated fat. T

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Factorable

Modelling Storm Surge Hazard to Mekong Deltating lipoprotein fractions, an individual’s CVD risk is also influenced by the concomitant presence or absence of other standard CVD risk factors. In this chapter, we describe the strengths and weaknesses of common circulating lipoproteins that are measured in clinical practice for predicting CVD ri

青春期

https://doi.org/10.1007/978-3-642-52438-7ong and internally consistent. Youth affected with dyslipidemia include those with single gene disorders such as familial hypercholesterolemia (FH), and those with oligogenic disorders such as familial combined hyperlipidemia (FCHL). In large populations of children followed up for several decades,

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Frank Fischer,Katharina Fischer. Dyslipidemia in type 2 diabetes is characterized by elevated levels of plasma total and very-low-density lipoprotein triglycerides and postprandial triglyceride-rich lipoprotein remnants, abundance of small dense low-density lipoprotein (LDL) particles, low levels of HDL cholesterol, and elevated

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畏缩

转向

Mumita Tanjeela,Md. Anisur RahmanUntreated HIV infection results in an initial fall of high-density lipoprotein (HDL) cholesterol, followed by declines in non-HDL cholesterol fractions. With progression to the acquired immune deficiency syndrome (AIDS), serum triglyceride levels rise. These changes appear principally driven by infl

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Peter Bühler,Patrick Schlaich,Dominik Sinnerlesterol and a high risk of premature cardiovascular disease (CVD). There are autosomal dominant and recessive MHs, although the latter are very rare. The most common and best studied MH is familial hypercholesterolemia (FH), an autosomal codominant disease caused by mutations in the LDL receptor ge

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