Titlebook: Drugs of Abuse, Immunity, and Immunodeficiency; Herman Friedman,Steven Specter,Thomas W. Klein Book 1991 The Editor(s) (if applicable) and

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Distillation Experiments and Observations, syndrome (AIDS) has stimulated a myriad of studies to identify cofactors capable of promoting HIV replication, thereby hastening the development of overt disease. One group of substances that has been touted as possible candidate cofactors in the progression of AIDS is drugs of abuse (1). The inabi

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Distillation Experiments and Observations,deficiency virus (HIV) infection (1,2). In the 1980s, HIV infection, with the resultant development of acquired immune deficiency syndrome (AIDS), became one of the leading causes of morbidity and mortality in parenteral drug abusers (3–5). Due to the long latency period of HIV infection, the number

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,The Flavor–Chemistry Association,he infectious disease complications of intravenous (IV) drug use were clearly recognized by the mid-20th century (2,3), serious attention to the mechanisms whereby opiates might participate in the pathogenesis of infectious disease did not surface until the past decade. Contemporary interest in this

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Cannabinoid Induced Changes in Eicosanoid Synthesis by Mouse Peritoneal Cells,e is not entirely understood, however, it has been suggested that it may have a role in macrophage function in the immune system. For some time we have utilized mouse peritoneal cells as a model system in which to study cannabinoid effects on the release of arachidonic acid and its subsequent conversion to certain eicosanoids (1).

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Effects of Cocaine and Other Drugs of Abuse on Immune Function,ymphocytes after mitogenic stimulation . with the plant lectin, phytohemag-glutinin. Later, Madden et al. (2) showed that this damage was probably due to altered DNA-repair capacity of lymphocytes. Less obvious, perhaps, than the genetic implications of these findings were their immunological implications, which, in fact, were significant.