Titlebook: Drugs and Central Synaptic Transmission; P. B. Bradley,B. N. Dhawan Textbook 1976Latest edition Macmillan Publishers Limited 1976 drugs.re

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Micronutrients and Other Trace Elements,amine pathways (Carlsson Rosengren, Bertler and Nilsson, 1957; Moore and Rech, 1967; Everett, 1961; Kadzielawa and Widy-Tyszkiewicz, 1970), in appreciation of the fact that .-DOPA is the natural precursor of the neurotransmitters dopamine and noradrenaline. Since the amino acid enters the catecholam

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Bacteria, Phytoplankton, and Water Quality, or a hallucinogen, although it has some properties in common with all these groups of drugs. The overall actions of Δ.-THC in brain are a mixture of apparent stimulatory effects with superimposed depressant manifestations, although there is considerable overlapping between these symptoms depending

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Dissolved Oxygen and Redox Potential,able interest has been focused on a possible relationship between cholinergic and dopaminergic systems in the central nervous system (CNS). In this study we investigated the effect of an anticholinergic drug, dexetimide (Janssen, Niemegeers, Schellekens, Demoen, Lenaerts, Van Nueten, Van Wijngaarden

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Pseudospectral and Galerkin Methodspounds which selectively antagonise transmitter action in the periphery are often used in the CNS in an attempt to block synaptic transmission. The selective nature of their action on the central neurones is open to considerable doubt however. Bevan, Bradshaw, Roberts and Szabadi (1974) found that m

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Role of putative central neurotransmitters in self-stimulation behaviour,eas of the brain through an implanted electrode. In the rat, such positive reinforcement has been demonstrated with respect to various brain areas extending from the forebrain to the rostral pons, most prominent among them being the medial forebrain bundle in the posterior lateral hypothalamus (Olds, 1958; 1962).

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