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Titlebook: Drugs Affecting Lipid Metabolism; Rodolfo Paoletti (Dean),David Kritchevsky (Associa Conference proceedings 1987 Springer-Verlag Berlin He

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Cellular and Molecular Biology of Apolipoproteins: Receptor-Mediated Regulation of Cholesterol MetaLipoprotein receptors, especially those within the liver, play a central role in the regulation of cholesterol metabolism. Specific apolipoproteins (apo-) serve as the ligands for these receptors (for review, see Brown and Goldstein 1983, 1986; Mahley and Innerarity 1983; Mahley et al. 1984).
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Lipolysis and Antilipolytic Drugs at Different Ages,Adipose tissue plays a major role in caloric homeostasis by shifting between lipid mobilization and storage. Its primary functions can be summarized as follows:
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0172-6625 opment of this area. After the publication and wide acceptance of the results of the cholestyramine study by the Lipid Clinics in the USA, showing for the first time a direct association between drug induced reduction of plasma levels of total and LDL cholesterol and coronary heart disease in a high
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Understanding Measures of Landscape Patternt, baby brain and intestine replicate at rapid rates and have active cholesterol synthesis. As we showed some years ago, the very rapid cell growth of regenerating liver is accompanied by one of the highest rates of cholesterolgenesis seen in mammalian cells (1).
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Keith Kroll,James Bassingthwaightenificantly decreased in different group populations including survivors of myocardial infarction, subjects with angina pectoris, and others with coronary artery disease as documented by angiography (1–4). In one study, apoA-I was found to be a superior predictor of CAD in individual patients compared to concentrations of HDL cholesterol (4,5)
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Keith Kroll,James BassingthwaighteL), lipoprotein lipase (LPL), and hepatic lipase (HL) represent important points of regulation in lipoprotein metabolism and are consequently major determinants for the concentrations of plasma-lipoproteins.
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APOA-I and APOA-II Metabolism and Coronary Artery Disease,nificantly decreased in different group populations including survivors of myocardial infarction, subjects with angina pectoris, and others with coronary artery disease as documented by angiography (1–4). In one study, apoA-I was found to be a superior predictor of CAD in individual patients compared to concentrations of HDL cholesterol (4,5)
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Lipolysis and Lipoprotein Metabolism,L), lipoprotein lipase (LPL), and hepatic lipase (HL) represent important points of regulation in lipoprotein metabolism and are consequently major determinants for the concentrations of plasma-lipoproteins.
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