Titlebook: Dopaminergic Systems and their Regulation; G. N. Woodruff (Director of Pharmacology),J. A. Po Book 1986 The Contributors 1986 dopamine.reg

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Autobiography

纹章

北极人

Schreibdidaktik und Schreibunterrichtoughout the brain. However, postmortem studies suggest that alterations also occur in other neuronal systems including those utilising noradrenaline, 5HT, GABA, acetylcholine and neuropeptides. The primary role played by the deficit in brain dopamine content is emphasised by the ability of L-DOPA or

使服水土

Thomas Nette,Jennifer Schell,Rolf Walterestigation. In its simplest form the hypothesis proposes that an increase in brain dopaminergic function may be responsible for the production of some of the symptoms of schizophrenia. Two major lines of evidence support the hypothesis. Firstly the dopamine receptor antagonism displayed by neurolept

META

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intrigue

Mechanisms Underlying the Modulation of Dopamine Release through Presynaptic Muscarinic Receptorsand the release of previously taken up .H-DA evoked by electrical stimulation from rabbit caudate slices (Helmreich et al., 1982; Lehmann and Langer, 1982) or by K. -depolarization from rat striatal synaptosomes (Raiteri et al., 1982) can be potentiated by acetylcholine (ACh) through the activation of receptors of the muscarinic type.

鲁莽

Pharmacological Characterization of the Dopamine Renal Vascular Receptor the autonomic nervous system, and particularly in the smooth muscle fibres of the renal vascular bed. This action is complex, involving postsynaptic alpha and beta-adrenoceptors as well as the dopamine receptors described as “postsynaptic”, although it has not yet been shown that dopamine is a neurotransmitter outside the central nervous system.

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