Titlebook: Dopamine in the CNS II; Gaetano Chiara Book 2002 Springer-Verlag Berlin Heidelberg 2002 Behavioural Psychology.Cell Biology.Central Nervou

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Sleep and Wake Cycle,ations by conduction transection experiments in cats. A preparation done in the intercollicular level (cerveau isolé) created a persistent slow-wave state in the midbrain, while an incision in the medulla oblongata (encephale isolé) did not alter the sleep-wake cycle. Because of this, he assumed tha

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Springer Fachmedien Wiesbaden GmbHe which control the rate of the transmitter synthesis are negatively coupled to adenylyl cyclase (. et al. 1985, 1986; . et al. 1988). In addition, these D. autoreceptors regulate the state of excitability of nerve terminal arborisations (. and . 1985; . et al. 1986).

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https://doi.org/10.1007/978-3-658-19628-8m has been more extensively studied because the loss of these dopaminergic neurons leads to Parkinson’s disease, and GABAergic neurons normally controlled by nigrostriatal dopamine are thought to play a critical role in the symptoms of the disease (see . and . 1996).

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,Dopamine — Acetylcholine Interactions,yric acid (GABA)ergic medium-size spiny neurons where they finally converge with DA neurons (. et al. 1994a). Acetylcholine, on the other hand, modulates the function of dopamine mesencephalic neurons by an action on nicotinic receptors by virtue of cholinergic projections from pontomesencephalic cell groups (. et al. 1999).

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,Dopamine — GABA Interactions,m has been more extensively studied because the loss of these dopaminergic neurons leads to Parkinson’s disease, and GABAergic neurons normally controlled by nigrostriatal dopamine are thought to play a critical role in the symptoms of the disease (see . and . 1996).