Titlebook: Dopamine and Sleep; Molecular, Functiona Jaime M. Monti,S. R. Pandi-Perumal,S. Chokroverty Book 2016 Springer International Publishing Swit

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Sleep in Schizophrenia Patients and the Effects of Second Generation Antipsychotic Drugs,TST), sleep efficiency (SE), and rapid-eye-movement (REM) sleep latency, but REM sleep percentage remain unchanged. According to polysomnographic studies, the administration of clozapine, olanzapine and paliperidone to schizophrenia patients was followed by a significant reduction of SL and an incre

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Sleep in Narcolepsy and the Effects of Modafinil, type 2), plus or minus hypnagogic hallucinations, sleep paralysis and disturbed nocturnal sleep. Narcolepsy type 1 is caused by a selective loss of hypocretin producing cells in the posterior hypothalamus. The pathogenesis is unknown but thought to be related to an autoimmune process, based on the

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Long-Term Management Problems in Restless Legs Syndrome (RLS)/Willis-Ekbom Disease (WED),de effects, which compromise the efficacy of treatment and force some patients to discontinue medication. This chapter addresses these long-term consequences to RLS treatment, which could be both disease-related and medication-related issues. In addition, this chapter briefly discusses how to manage

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Dopaminergic Transmission and Wake-Promoting Effects of Central Nervous System Stimulants,se arousal by inhibiting the nucleus accumbens, by exciting wake-promoting basal forebrain cholinergic and brainstem serotonin neurons and by inhibiting sleep-promoting neurons in the preoptic hypothalamus. Dopamine acts on D. type (D., D.) and D.-type (D., D., D.) receptors. Both types are involved

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Functional Interactions Between MCHergic and Dopaminergic Neurons: Role in the Control of Wakefulneing non-REM (NREM) sleep and reach their maximal rate during REM sleep. Although there is almost no direct information regarding the interaction between MCHergic and DAergic neurons in the control of W and sleep, indirect evidence strongly suggests an important interaction between both systems in th

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Alteration of Biological Rhythms in Diseases of the Central Dopaminergic System: Focus on Parkinsond SCN, these pieces of evidence suggest that disturbances in the circadian timing system might be part of the core features of PD and not just a “collateral damage”. According to this view, a disturbed clockwork might actively contribute to neurodegeneration and a chronotherapeutic approach to PD mi

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Depression, Sleep Disorders, and DA,nown. The evidence implicates dopamine as a neurobiological factor associated with symptoms of insomnia and depression. Dopamine is a neuromodulator that regulates reward processing, arousal states, affect, and mood regulation. However, the putative role of dopamine in insomnia and depression has la