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Titlebook: Diseases in the Elderly; Age-Related Changes Nages Nagaratnam,Kujan Nagaratnam,Gary Cheuk Book 2016 Springer International Publishing Swit

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Low and Loss of Vision in the Elderly,ath of the retinal ganglion cells causing irreversible visual loss. Cataract of old age is multifactorial with genetic, environmental and biochemical factors which act synergistically. A number of biochemical pathways have been proposed as possible links between hyperglycaemia and diabetic retinopat
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Connective Tissue Disorders and Vasculitis in the Elderly,ved in the vascular inflammation, namely, immune-complex disease, antibody- mediating disease, antibody-dependent cellular toxicity, and endothelial activation among others. The involvement of different end organs, the kidney, lung, central nervous system and gastrointestinal tract, and the spectrum
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Shidi Dong,Lei Xu,Ron McIver,Ning Huangulus resulting in aortic sclerosis and mitral incompetence and intimal hyperplasia and thickening of the arteries leading to an increase in systolic pressure and systolic hypertension. The electrical characteristics of the conducting system also change with age, and the time of conduction through th
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The Real Discovery: The Web is Glial,, autoimmune hepatitis(AIH), drug-induced hepatitis(DILI), primary biliary cirrhosis (PBC) and hereditary haemochromatosis (HH). The exact pathogenesis of alcoholic liver disease is complex and implicates alcoholic metabolism and secondary mechanisms. A genetic susceptibility as predisposing factor
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The Real Discovery: The Web is Glial, Many old people suffer from vitamin B. deficiency. There are three groups of haematological neoplastic disorders in the elderly and increasing in incidence with advancing age, namely, myelodysplasia and acute myeloid leukaemia, multiple myeloma and chronic lymphatic leukaemia. The chronic myeloprol
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Getting There: Landing an Onion on Mars,uding acute glomerulonephritis, interstitial nephritis, acute tubular necrosis and disease of the renal arteries. In chronic kidney disease (CKD), there is a progressive loss of nephrons irrespective of the primary site of the insult, cortical or medullary whether resulting from glomerular, tubular
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