Titlebook: Diastolic Relaxation of the Heart; The Biology of Diast Beverly H. Lorell (Associate Professor of Medicine Book 1994Latest edition Springer

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Molecular Physiology of Ventricular Hypertrophye of the pathophysiologic stimulus, the hypertrophic response of the myocardium can assume many forms, shapes, and sizes, leading to concentric hypertrophy, dilated hypertrophy, asymmetric hypertrophy, and enlargement of one to four chambers. While the global structure of the myocardium varies widel

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Cardiac Renin-Angiotensin System in Cardiac Hypertrophy and Failurewalls with minimal enlargement of the left ventricular cavity. In addition to the overall stimulation of cardiac muscle growth, pressure overload induces a fetal pattern of gene programming, including preferential synthesis of β-myosin heavy chain and α-skeletal actin isoforms, which promote a slowe

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Effects of Hypoxia and Ischemia on Intracellular Calcium and Relaxation: Studies in the Aequorin-Loamic heart disease affects some 7 million Americans, with an estimated annual economic impact of $43 billion, and remains the leading cause of death in the United States [1]. Both hypoxia/reoxygenation and ischemia/ reperfusion are associated with marked abnormalities of cardiac contraction and rhyth

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Diastolic Dysfunction During Ischemia: Role of Glycolytic ATP Generationl Ischemia.” Opie reasoned that “glycolysis may be good for the survival of ischemic myocardium. . . Increasing blood glucose concentrations may benefit the ischemic myocardium by increasing the rate of anaerobic glycolysis, by reversing ion losses, by a direct membrane effect, by altering the extra