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Titlebook: Diagnosis and Therapy of Porphyrias and Lead Intoxication; International Sympos Manfred Doss Conference proceedings 1978 Springer-Verlag Be

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发表于 2025-3-21 16:39:43 | 显示全部楼层 |阅读模式
书目名称Diagnosis and Therapy of Porphyrias and Lead Intoxication
副标题International Sympos
编辑Manfred Doss
视频video
图书封面Titlebook: Diagnosis and Therapy of Porphyrias and Lead Intoxication; International Sympos Manfred Doss Conference proceedings 1978 Springer-Verlag Be
描述On the occasion of the 450th anniversary of Philipp University, the porphyrin research group of. the Department of Clinical Biochemistry in the Faculty of Medicine organized an international symposium. This was held between June 28 and July 1, 1977, under the joint chairman­ ship of Professor Samuel Schwartz and the editor. The organizers and chairmen of the symposium wish to thank the Sozialminister of Hessen, Armin Clauss, the President of the German Society for Clinical Chemis­ try, Professor A. Delbrlick, and the Dean of the Faculty of Medicine of Philipp University, Professor F. Neurath, for their support and patronage. We are also grateful to Professor A. Treibs of the Institute of Organic Chemistry, Technical University of Munich. Professor Treibs, a former pupil, co-worker and now successor to the Nobel laureate Hans Fischer, kindly accepted the honorary presidency of this international symposium. The opening ceremony was inaugurated by the chairmen, with welcoming speeches by the President of Philipps University, Mr. Rudolf Zingel and patrons of the university. The editor outlined the 10 years of development of clinical biochemistry at Philipp University and traced in deta
出版日期Conference proceedings 1978
关键词absorption; anemia; biochemistry; carcinoma; chemistry; diagnosis; diagnostics; differential diagnosis; embr
版次1
doihttps://doi.org/10.1007/978-3-642-67002-2
isbn_softcover978-3-540-08863-9
isbn_ebook978-3-642-67002-2
copyrightSpringer-Verlag Berlin Heidelberg 1978
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Differential Patterns of Heme and Prophyrin Biosynthesis in Liver and Kidney: Metal Ion Blockade of r the most part, is due to the ready induction response of liver δ-aminolevulinate synthetase (ALAS) — the rate-limiting enzyme of porphyrin-heme synthesis — to the exogenous chemicals and the physiologic factors that alter porphyrin and heme production. Compounds known to be highly potent in alteri
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Permissive Effects of Hormones on the Induction of δ-Aminolevulinic Acid Synthase in Cultured Chick-s enzyme is present in the mitochondria and is inducible in the liver by a variety of compounds of hydrophobic nature. Studies of the regulation of ALA-S have been facilitated by the introduction of a tissue-culture technique for cultivating monolayer of chick-embryo liver cells (2). A major drawbac
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A New Approach to Porphyriaine, has been very helpful for their diagnosis (8). Consecutive biochemical and clinical findings subdivided each group: erythropoietic porphyrias on the basis of different porphyrins occurring in the blood, hepatic porphyrias on the basis of porphyrins and precursors in urine, feces, and liver (8)
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The Clinical Chemistry of Variegate Porphyria with Special Reference to the Identification of a New (8), with an estimated incidence of approximately 9000 affected persons. How this remarkable situation has come about has been fully documented by Dean (5) in his monograph, which makes it clear that virtually all South African variegate prophyrics are descendants of a daughter of a marriage that t
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Expression of the Gene Defects of Acute Intermittent Porphyria (AIP) and Erythropoietic Protoporphyric cells (7) reflects the primary gene defect of AIP, and accounts for the excessive urinary excretion of porphobilinogen and 6-aminolevulinic acid (ALA). The decreased activity of ferrochelatase found in cultured skin fibroblasts and bone-marrow cells from patients with erythropoietic protoporphyri
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A Case of Acute Intermittent Porphyria, Relapsing Acute Pancreatitis, and Unconjugated Hyperbilirubi The previously healthy 36-year-old woman was admitted to the hospital because of severe colicky pain in the right lower abdomen, vomiting, constipation, and red colored urine. A highly increased excretion of porphobilinogen in the urine and a normal fecal excretion of porphyrins was compatible with
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