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Titlebook: Diabetic Renal-Retinal Syndrome; 21st Century Managem Eli A. Friedman (Distinguished Teaching Professor) Book 1998 Springer Science+Busines

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Mathias Berek,Kristina Chmelar,Gerd Sebaldal impression that cigarette smoking is deleterious and actually hastens the onset of uremia in the type 2 patient. Pointing to cluster analysis in family studies, the authors suggest that genetic predetermination for nephropathy is coded separately from the risk of type 2 diabetes. The references c
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https://doi.org/10.1007/978-3-658-26593-9n are reduced. The utility of ranking extrarenal diabetic complications numerically using a Co-Morbidity Index has been demonstrated in studies of hemodialysis and peritoneal dialysis patients and is proposed for kidney transplant recipients.
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,Der Begriff „soziale Gedächtnisse“,y trials of dialytic therapy in diabetes when first-year survival was less than 25%, current prospects for a 75% survival in newly started hemodialysis patients indicates remarkable progress. Still, rehabilitation presently is far from satisfactory and further modifications in diabetic management to
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https://doi.org/10.1007/978-3-658-26593-9ic graft over a primary arterio-venous fistula is often a decision forced by inadequate or damaged vessels at the time of access creation. No easy remedy for effective vascular access in diabetic hemodialysis patients is evident.
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Der Umgang mit dem Klassenfeindtiblindness technique. Every diabetic person referred for a renal evaluation must be seen by a collaborating ophthalmologist early and repeatedly as a component of state-of-the-art comprehensive management. Burning the retina to preserve sight though counterintuitive is nevertheless an essential int
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Friedemann Geisler,Friedel Geislerpermeability by modifying cellular tight junction proteins of which ZO-1 has been shown to be regulated by histamine. Further, VEGF reduces the amount of occludin, another seven junctional proteins that spans plasma membranes serving as a transmembrane protein permitting cell-to-cell interaction thr
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https://doi.org/10.1007/978-3-531-91974-4itreous and aqueous fluid samples of patients with active proliferative diabetic retinopathy but low in control patients and in diabetic subjects without retinopathy. A fascinating and promising approach, based on altering the molecular biology of retinal cells, to blunting proliferative diabetic re
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