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Titlebook: Developmental Neuropathology of Schizophrenia; Sarnoff A. Mednick,Tyrone D. Cannon,José M. LaFoss Book 1991 Springer Science+Business Medi

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发表于 2025-3-21 18:33:41 | 显示全部楼层 |阅读模式
书目名称Developmental Neuropathology of Schizophrenia
编辑Sarnoff A. Mednick,Tyrone D. Cannon,José M. LaFoss
视频video
丛书名称NATO Science Series A:
图书封面Titlebook: Developmental Neuropathology of Schizophrenia;  Sarnoff A. Mednick,Tyrone D. Cannon,José M. LaFoss Book 1991 Springer Science+Business Medi
描述This volume reports the proceedings of a NATO Advanced Workshop held in Castelvecchio Pas coli, Italy, from August 28 - September 1, 1989. An important inspiration for this Workshop came from our studies in Helsinki and Denmark, which have found that exposure to an influenza epidemic during the second trimester of fetal development increases the risk of adult schizophrenia. This finding has stimulated an important new hypothesis in the study of the etiology of schizophrenia. It has suggested the possiblity that disturbances of brain development during gestation may contribute to the risk of adult schizophrenia. We determined that it would be of value to bring together schizophrenia researchers and those doing basic studies of the development of the brain. Both groups of researchers were encouraged to communicate at a level that would help other scientists to integrate their knowledge and techniques into their own discipline. For this reason, perhaps, the papers of this volume are remarkably clear and not difficult to understand. The first four chapters describe the neurochemical and structural aspects of brain development. The chapter by Dziegielewska and Saunders discusses transpo
出版日期Book 1991
关键词Neuron; brain; cerebral cortex; cortex; development; dopamine; magnetic resonance imaging (MRI); neuropatho
版次1
doihttps://doi.org/10.1007/978-1-4615-3378-8
isbn_softcover978-1-4613-6491-7
isbn_ebook978-1-4615-3378-8
copyrightSpringer Science+Business Media New York 1991
The information of publication is updating

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Nicholas Polys,Peter Sforza,John Munsellstinguishable developmental courses and psychopathologic evolutions. An outline of these hypothetical paths to schizophrenia is presented as a heuristic for understanding heterogeneity of the cerebral morphological, neuropsychological, and clinical features of the schizophrenic syndrome.
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Opportunities in Conflict on the Trails such as computed tomography (CT), magnetic resonance imaging (MRI), positron emission tomography (PET), single photon emission computed tomography (SPECT), and regional cerebral blood flow (rCBF) studies as well as from new morphometric post mortem methods.
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PlantShoe: Botanical Detectives published examining links between genetic predisposition, obstetrical complications, and schizophrenia. This chapter examines three etiological models which include obstetrical complications as a factor.
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https://doi.org/10.1007/978-3-031-02197-8n et al., 1956). Although these investigations have in some cases enlightened us to the impact of family variables (Brown et al., 1986; Leff, 1976), they have for the most part not proven sufficient to explain the development of the disorder.
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Is the Differential Localization of Specific Plasma Proteins in the Developing Neo- and Archicortex capillaries, epithelial cells in choroid plexuses and arachnoidal cells in meninges, provide the underlying diffusion restraint of the brain barriers upon which all other mechanisms act (see Dziegielewska & Saunders, this volume).
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Obstetrical Events and Adult Schizophrenia published examining links between genetic predisposition, obstetrical complications, and schizophrenia. This chapter examines three etiological models which include obstetrical complications as a factor.
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