Titlebook: Developmental Neuropathology; Reinhard L. Friede Book 19751st edition Springer-Verlag Wien 1975 neuropathology

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indigenous

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Hemorrhages Characteristic of Asphyxiated Premature Infantsin of asphyxiated premature infants. The incidence of intraventricular hemorrhage has been reported at approximately 1.1 per 1,000 live births (Fedrick and Butler, 1970), but the figure is of limited significance as some degree of subependymal bleeding is extremely common in the newborn succumbing t

rectum

Cerebral Lesions from Physical Traumamatures are mainly subependymal and intraventricular, leptomeningeal, or in the cerebellar parenchyma, and are related to asphyxia. They become less frequent as the infant approaches maturity. The types of hemorrhages characteristic of the mature newborn are related to mechanical trauma, such as lac

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ALIAS

Diffuse Fatty Change of White Matteristing of this tissue alteration among the lesions of asphyctic and traumatic birth is open to challenge. Its description at this point of the text is merelya matter of concern for its overlap with the periventricular infarcts described in Chapter 4. Virchow (1867) described diffuse fatty change of

Vaginismus

Perinatal Lesions of Cerebral and Cerebellar Cortex the brain. In the first case there was a cleft in the region of the left Sylvian fissure, communicating with the ventricle, and small convoluted gyri were abundant in both frontal and anterior parietal lobes. The second showed foci of small gyri in approximately symmetrical distribution, parasagitt

pacific

Lesions of Basal Ganglia, Brain Stem and Cordpaced irregular zones of gray matter were found in the posterior halves of both putamina. Microscopic examination showed scar tissue containing myelinated nerve fibers. Anton concluded that the lesions were residual to vascular softenings dating to the first year of life, and were the anatomical sub

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Kernicterus (Bilirubin Encephalopathy)perficial portions of brain tissue in the ventricular walls and at the hemispheric surface. The first comprehensive description of the disease was given by Schmorl (1903) who distinguished two types of cerebral lesions in icteric infants. One type consisted of a diffuse staining of the tissue and wa

isotope

Various Topographic Patterns of Postnatal Neuron Lossgic and pathogenetic mechanisms are the same. The lesions are discussed in separate chapters, however, because of differences in the onset of the underlying disease processes in relation to birth. The lesions described earlier (Chapters 6, 7), such as ulegyria, marbled state, or pontosubicular neuro