Titlebook: Developmental Neuropathology; Reinhard L. Friede Book 1989Latest edition Springer-Verlag Berlin Heidelberg 1989 Hydrocephalus.aneurysm.bra

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Min-Gu Heo,Daehyoun Ki,Changhoon Parkersely, cardiac arrest encephalopathy, described in this chapter, was often reported as a typical form of perinatal damage. None the less, on review one will find that the lesions described in this chapter are more likely to be of postnatal origin and that they rarely or never arise before birth.

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Predicting Gender via Eye Movements thrombosis of intracranial sinus or veins, and five had abscesses. Various lesions were found in eight of the 27 acyanotic patients. Large series were published by Terplan (1973) and Bozóky et al. (1984).

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Asad Khan,Sunzhe Yang,Ian GonsherWatson (1957), Ziai and Haggerty (1958), Groover et al. (1961), Yu and Grauaug (1963), Overall (1970), and others. Recent symposia on neonatal infections were edited by Remington and Klein (1976) and Isacsohn (1983).

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Book 1989Latest editionook on pathology, and yet it witnessed many important changes of concepts, along with a formidable growth of knowledge. The second edition required extensive reorganization. There are new chapters on mitochondriopathies, on peroxisomal diseases and on spongy myelino­ pathies. Major revisions and new

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Perinatal Lesions of Gray Matterinatal lesions of the white matter. All of these lesions may coexist or combine in various patterns. The subsequent Chap. 8 reviews lesions which are more likely to result from disturbances after birth or during postnatal life. The distinction is only relative, however, as there is generous overlap between these groups.

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Purulent Leptomeningitis in Newborns and InfantsWatson (1957), Ziai and Haggerty (1958), Groover et al. (1961), Yu and Grauaug (1963), Overall (1970), and others. Recent symposia on neonatal infections were edited by Remington and Klein (1976) and Isacsohn (1983).

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